The higher ratio of placental pathologic findings in patients with infantile hemangioma suggests that reduced placental oxygen diffusive conductance contributes to fetal hypoxic stress and that hypoxic/ ischemic changes in the placenta could be related to infantile hemangioma development via vascular endothelial growth factor and placental growth factor expression, among others, within the villious …

2021-03-28

Design and Patients Twelve tissue specimens taken from infantile hemangiomas on patients aged 5 days to 2 years were retrospectively confirmed clinically and histologically. It has been reported that infantile hemangiomas have been described to occur more frequently in association with chorangiomas (a type of hemangioma), which is the most frequently occurring benign tumor of the maternal placental trophoblasts . It reveals that there might be some correlation between infantile hemangioma and placental trophoblast. Various theories regrarding pathogenesis of hemangioma:-The placental theory is attractive because it would explain the programmed life cycle of infantile hemangioma (IH). This theory is based on evidence suggesting that the placenta is closely related to IH. As suggested from Hoeger, the association between hemangiomatosis and chorangioma supports a link between infantile hemangiomas and embolized placental cells, at least in cases of large chorangiomas, such as those observed in his study . Discuss theories that may account for hemangioma-genesis.

Infantile hemangioma placenta theory

Special emphasis will be placed on placental This review focuses on the placental theory, which proposes that a fetal placental progenitor is the cell type of origin for infantile hemangioma. Special emphasis will be placed on placental vasculogenesis and the presence and transit of placental progenitor cells during gestation. Background: The unique immunobiology of the placental trophoblast and the increased incidence of hemangiomas in infants born after chorionic villus sampling suggest that an immunologically regulated ectopic focus of trophoblasts could be the cell of origin for proliferative infantile hemangiomas. The higher ratio of placental pathologic findings in patients with infantile hemangioma suggests that reduced placental oxygen diffusive conductance contributes to fetal hypoxic stress and that hypoxic/ischemic changes in the placenta could be related to infantile hemangioma development via vascular endothelial growth factor and placental growth factor expression, among others, within the villious vessels and throphoblasts. 2001-05-01 The higher ratio of placental pathologic findings in patients with infantile hemangioma suggests that reduced placental oxygen diffusive conductance contributes to fetal hypoxic stress and that hypoxic/ ischemic changes in the placenta could be related to infantile hemangioma development via vascular endothelial growth factor and placental growth factor expression, among others, within the villious … Abstract. The relationship between infantile hemangioma (IH) and the placenta has been largely discussed in the last 5 years ( 1 ). North et al documented in 2001 the expression of placental vascular epitopes in hemangiomas, suggesting the origin of these tumors from placental endothelial cells or their precursors ( 2–4 ).

Shared expression of distinct endothelial markers in hemangioma and placental tissues raises a possibility that infantile hemangioma is originated from placental trophoblast. Moreover, the findings of a very high similarity between the transcriptomes of placenta and hemangioma provide strong support for this theory.

Special emphasis will be placed on placental The higher ratio of placental pathologic findings in patients with infantile hemangioma suggests that reduced placental oxygen diffusive conductance contributes to fetal hypoxic stress and that hypoxic/ischemic changes in the placenta could be related to infantile hemangioma development via vascular endothelial growth factor and placental growth factor expression, among others, within the villious vessels and throphoblasts. Abstract. Background: The unique immunobiology of the placental trophoblast and the increased incidence of hemangiomas in infants born after chorionic villus sampling suggest that an immunologically regulated ectopic focus of trophoblasts could be the cell of origin for proliferative infantile hemangiomas.

We know that they are not inherited and, unlike infantile haemangiomas, they affect males and females equally. More research is needed to confirm the causes of congenital haemangiomas. One theory is that it is related to placental tissue. The placenta is divided into segments, similar to a flattened orange which has pith separating each segment.

Extrinsic theory suggests that external environmental factors provide an environment favourable for the development of infantile haemangioma. An infantile hemangioma (IH) is a type of benign vascular tumor that affects babies. They appear as a red or blue raised lesion.

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This theory the investigators noted a threefold increase in pathologic changes suggestive of hypoxia in the placenta of infants with hemangioma The placenta releases angiostatic factors (sFLT1), but after birth these inhibitory factors are wasted, which allows the development of the infantile hemangioma by endothelial cell proliferation Certain aspects of the biology of infantile hemangioma cells suggest a relationship to the placenta as a possible site of origin for the hemangioma precursor cells. In this article, a relationship between the placenta, with or without a chorangioma and the hemangioma sites of localization, is hypothesized. Se hela listan på emedicine.medscape.com Abstract. The relationship between infantile hemangioma (IH) and the placenta has been largely discussed in the last 5 years ( 1 ).

If the hypothesis proves to be valid, clues for possible treatment are outlined. Mihm MC, Nelson JS. Hypothesis: the metastatic niche theory can elucidate infantile hemangioma development An airway The placenta releases angiostatic factors (sFLT1), but after hemangioma is accompanied by feeding dificulty, birth these inhibitory factors are wasted, which allows the stridor, loud breathing and a typical cry, all of them development of the infantile hemangioma by endothelial representing signs of airway obstruction [19].
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of human placenta and infantile hemangioma are sufficiently similar to suggest a placental origin for this tumor, expanding on recent immunophenotypical studies that have suggested this pos-sibility [North, P. E., et al. (2001) Arch. Dermatol. 137, 559-570]. The transcriptomes of placenta, hemangioma, and …

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2014-09-01 · This theory is based on evidence suggesting that the placenta is closely related to IH. As suggested from Hoeger, the association between hemangiomatosis and chorangioma supports a link between infantile hemangiomas and embolized placental cells, at least in cases of large chorangiomas, such as those observed in his study [24] . Barnés CMChristison-Lagay EAFolkman J The placenta theory and the origin of infantile hemangioma. Lymphat Res Biol 2007;5 (4) 245- 255 PubMed Google Scholar Crossref 17. 2021-03-28 · The placenta theory and the origin of infantile hemangioma.

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This review focuses on the placental theory, which proposes that a fetal placental progenitor is the cell type of origin for infantile hemangioma. Special emphasis will be placed on placental vasculogenesis and the presence and transit of placental progenitor cells during gestation. This review focuses on the placental theory, which proposes that a fetal placental progenitor is the cell type of origin for infantile hemangioma. Special emphasis will be placed on placental The higher ratio of placental pathologic findings in patients with infantile hemangioma suggests that reduced placental oxygen diffusive conductance contributes to fetal hypoxic stress and that hypoxic/ischemic changes in the placenta could be related to infantile hemangioma development via vascular endothelial growth factor and placental growth factor expression, among others, within the villious vessels and throphoblasts. Abstract. Background: The unique immunobiology of the placental trophoblast and the increased incidence of hemangiomas in infants born after chorionic villus sampling suggest that an immunologically regulated ectopic focus of trophoblasts could be the cell of origin for proliferative infantile hemangiomas.